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Pediatric Hip Fractures: Diagnosis, Classification & Treatment

Last Revision May , 2026
Reading Time 6 Min
Readers 178 Times

Pediatric hip fractures are rare but potentially devastating injuries in children. They account for less than 1% of all fractures in the pediatric population and occur far less frequently than hip fractures in adults. Despite their rarity, these injuries are associated with serious complications, particularly osteonecrosis of the femoral head, premature physeal closure, coxa vara, and nonunion.

Most pediatric hip fractures result from high-energy trauma such as motor vehicle accidents or falls from significant heights. Prompt diagnosis and early anatomic reduction are critical to preserving blood supply to the femoral head and minimizing long-term disability.


Epidemiology

  • Pediatric hip fractures represent less than 1% of childhood fractures
  • Approximately 75%–80% are caused by severe trauma
  • More common in:
    • Motor vehicle collisions
    • Falls from height
    • Sports trauma
  • Pathologic fractures may occur through:
    • Bone cysts
    • Fibrous dysplasia
    • Bone tumors
  • Stress fractures are uncommon in children

Anatomy of the Pediatric Hip

Understanding proximal femoral anatomy is essential because growth plate injury and vascular disruption strongly influence prognosis.

Ossification Centers

  • Proximal femur begins ossification at approximately 7 weeks in utero
  • Proximal femoral epiphysis appears between 4–8 months of age
  • Greater trochanter ossifies around 4 years
  • Proximal femoral epiphysis fuses at approximately 18 years
  • Trochanteric apophysis fuses between 16–18 years

Blood Supply of the Femoral Head

The blood supply is primarily derived from:

  • Medial femoral circumflex artery (most important)
  • Lateral femoral circumflex artery

By age 3–4 years, the posterosuperior retinacular vessels become the dominant blood supply to the femoral head.

The artery of the ligamentum teres contributes minimally before age 8 years.

Disruption of these vessels significantly increases the risk of avascular necrosis (AVN).


Mechanism of Injury

Pediatric hip fractures may result from:

  • Axial loading
  • Torsional forces
  • Hyperabduction injuries
  • Direct trauma

Common mechanisms include:

  • Motor vehicle accidents
  • Pedestrian trauma
  • Falls from height
  • Sports injuries

Pathologic fractures should raise suspicion for underlying bone disease.


Clinical Presentation

Children with hip fractures commonly present with:

  • Severe hip or groin pain
  • Inability to bear weight
  • Shortened lower extremity
  • External rotation deformity
  • Painful hip motion
  • Swelling and ecchymosis
  • Tenderness around the proximal femur

A complete neurovascular examination is mandatory.


Diagnostic Evaluation

Plain Radiographs

Initial imaging includes:

  • Anteroposterior (AP) pelvis radiograph
  • Cross-table lateral hip radiograph

Internal rotation views may improve visualization if tolerated.

Computed Tomography (CT)

Useful for:

  • Nondisplaced fractures
  • Stress fractures
  • Preoperative planning

Magnetic Resonance Imaging (MRI)

MRI can detect occult fractures within 24 hours and evaluate associated soft tissue injury.

Bone Scan

Radioisotope bone scans may demonstrate increased uptake approximately 48 hours after injury.

Pediatric Hip Fracture xray

Delbet Classification of Pediatric Hip Fractures

The Delbet classification is the most widely used system and correlates strongly with the risk of osteonecrosis.

Type I: Transepiphyseal Fracture

  • Approximately 8% of pediatric hip fractures
  • Highest risk of osteonecrosis
  • Often associated with hip dislocation

Subtypes

  • Type IA: Without dislocation
  • Type IB: With dislocation

Key Features

Type II: Transcervical Fracture

  • Most common type
  • Represents approximately 45% of pediatric hip fractures
  • Frequently displaced

Complications

  • Osteonecrosis in up to 50% of cases

Type III: Cervicotrochanteric Fracture

  • About 30% of pediatric hip fractures
  • More common in children than adults

Complications

  • AVN rate approximately 20%–30%

Type IV: Intertrochanteric Fracture

  • Represents 10%–15% of cases
  • Lowest risk of AVN due to better blood supply
Pediatric Hip Fracture Delbet classification

Treatment of Pediatric Hip Fractures

Management depends on fracture type, displacement, age, and skeletal maturity.

General Principles

  • Urgent reduction and fixation
  • Restoration of anatomy
  • Preservation of femoral head blood supply
  • Stable fixation
  • Avoidance of physeal injury when possible

Type I Fracture Treatment

Recommended Management

  • Closed reduction and pin fixation
  • Smooth pins in younger children
  • Partially threaded pins in older children

Open reduction is indicated if closed reduction fails.

Type II Fracture Treatment

Nondisplaced Fractures

Options include:

  • Abduction spica cast
  • In situ pinning

Displaced Fractures

  • Closed reduction and internal fixation
  • Open reduction if necessary

Avoid transphyseal pin placement whenever possible.

Type III Fracture Treatment

Nondisplaced

  • Traction followed by spica casting
  • Immediate spica casting
  • In situ fixation

Displaced

  • Open reduction and internal fixation (ORIF)

Type IV Fracture Treatment

Nondisplaced

  • Traction for 2–3 weeks
  • Abduction spica cast for 6–12 weeks

Displaced or Unstable

  • Open reduction and internal fixation
Pediatric Hip Fracture fixation

Complications of Pediatric Hip Fractures

Osteonecrosis (Avascular Necrosis)

AVN is the most serious complication.

  • Overall incidence approximately 40%
  • Risk increases with:
    • Proximal fracture location
    • Fracture displacement
    • Delay in treatment

Ratliff Classification of AVN

Type I
  • Complete femoral head involvement
  • Femoral head collapse
  • Poor prognosis
Type II
  • Partial head involvement
  • Minimal collapse
Type III
  • Femoral neck involvement only
  • Femoral head spared

Premature Physeal Closure

Occurs in up to 60% of cases.

Potential consequences include:

  • Femoral shortening
  • Coxa vara
  • Short femoral neck
  • Limb length discrepancy

Risk increases with transphyseal fixation.

Coxa Vara

Occurs in approximately 20% of patients.

Usually caused by:

  • Inadequate reduction
  • Loss of fixation
  • Malunion

Anatomic reduction and stable fixation reduce incidence.

Nonunion

Occurs in approximately 10% of cases.

Risk factors include:

  • Poor reduction
  • Inadequate fixation
  • Delayed diagnosis

Treatment may require:

  • Valgus osteotomy
  • Bone grafting
  • Revision fixation

Prognosis

Outcomes depend on:

  • Fracture type
  • Degree of displacement
  • Time to treatment
  • Presence of osteonecrosis

Type I fractures have the worst prognosis because of the extremely high risk of femoral head ischemia.

Early diagnosis and stable fixation significantly improve long-term outcomes.


Key Points

  • Pediatric hip fractures are rare but serious injuries
  • Most result from high-energy trauma
  • Delbet classification predicts AVN risk
  • Type I fractures have the highest complication rate
  • Urgent reduction and fixation are critical
  • Osteonecrosis remains the most feared complication
  • Long-term follow-up is essential to monitor growth disturbance and deformity

Frequently Asked Questions (FAQs)

What is the most common pediatric hip fracture?

Delbet Type II transcervical fractures are the most common pediatric hip fractures.

What is the most serious complication?

Avascular necrosis (osteonecrosis) of the femoral head is the most serious complication.

Why are pediatric hip fractures dangerous?

These fractures may disrupt blood supply to the femoral head, causing permanent deformity and arthritis.

Can children recover fully from hip fractures?

Many children recover well with prompt treatment, but complications such as AVN or growth disturbance can affect long-term outcomes.


Conclusion

Pediatric hip fractures are uncommon orthopedic emergencies associated with substantial morbidity. Accurate diagnosis, understanding of proximal femoral vascular anatomy, and prompt surgical management are essential to reduce complications and optimize outcomes. The Delbet classification remains the cornerstone for prognostication and treatment planning. Long-term surveillance is critical because growth disturbance and osteonecrosis may develop months or years after injury.


References & More

  1. Boardman MJ, Herman MJ, Buck B, Pizzutillo PD. Hip fractures in children. J Am Acad Orthop Surg. 2009 Mar;17(3):162-73. doi: 10.5435/00124635-200903000-00005. PMID: 19264709. Pubmed
  2. Theunissen WWES, Veldman HD, Witlox MA. Een heupfractuur bij een kind [Management of pediatric hip fractures]. Ned Tijdschr Geneeskd. 2025 Aug 11;169:D8458. Dutch. PMID: 40831414. Pubmed
  3. Papalia R, Torre G, Maffulli N, Denaro V. Hip fractures in children and adolescents. Br Med Bull. 2019 Mar 1;129(1):117-128. doi: 10.1093/bmb/ldz004. PMID: 30753305. Pubmed
  4. Lark RK, Dial BL, Alman BA. Complications After Pediatric Hip Fractures: Evaluation and Management. J Am Acad Orthop Surg. 2020 Jan 1;28(1):10-19. doi: 10.5435/JAAOS-D-17-00689. PMID: 31633660. Pubmed
  5. Egol KA. Handbook of fractures. 6th ed. Philadelphia: Lippincott Williams & Wilkins; 2019.

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