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Thomas Test

 Thomas Test

Thomas Test (or as it called Hugh Owen Thomas well leg raising test) is used to measure the flexibility of the hip flexor muscles. It’s a test for hip flexor tightness.

Thomas Test is used to evaluate hip flexion contracture and psoas syndrome (Iliopsoas Tightness), which is more common in runners, dancers, and gymnasts with symptoms of hip “stiffness” and “clicking” feeling when flexing at the waist.

The original Thomas test was designed to test the flexibility of the iliopsoas complex but has since been modified and expanded to assess a number of other soft tissue structures.

See Also: Hip Muscles Anatomy

The hip flexor muscles are (See Below):

  1. The iliopsoas muscle group (It’s made up of 3 muscles, the Psoas Major, Psoas Minor and Iliacus muscle).
  2. The rectus femoris muscle.
  3. Pectineus muscle.
  4. Gracillis muscle.
  5. Tensor fascia latae muscle.
  6. Sartorius muscle.

Thomas test was first described by Dr. Hugh Owen Thomas, a British orthopedic surgeon (1834–1891). He is considered the father of orthopedic surgery in Britain.

See Also:  Hamstring Flexibility Test

How do you perform the Thomas Test?

Prerequisites for thomas test:

  1. Hard fat surface (on a sofa couch exaggerated lordosis and its obliteration are not well appreciated).
  2. Sufficiently undressed patient in a well-illuminated room to visualize the lumbar lordosis and ischial tuberosity.

The original Thomas test of the hip involves positioning the patient in supine, with one knee being held to the chest at the point where the lumbar spine is felt to flex. The clinician assesses whether the thigh of the extended leg maintains full contact with the surface of the bed.

Another way to do Thomas test: With patient supine with both hips flexed and maintaining one hip in flexion (to keep the pelvis fixed in corrected position),the patient is asked to actively extend the limb as much as he/she can. Thomas test Positive if unable to touch posterior thigh with examination table.

The angle between the thigh and the hard surface gives an idea of the flexion contracture at the hip.

An increased flexion contracture in the hip can be compensated for by an increase in lumbar lordosis, in which case the patient only appears to assume a normal position.

What does a positive Thomas Test mean?

One of the limitations of thomas test is that it merely determines the amount of hip extension possible at any given degree of pelvic flexion. Another problem is that there are better methods of measuring the flexibility of the iliopsoas complex. For example, positioning the patient in prone, stabilizing the pelvis, and then extending the thigh. The precise point at which the pelvis begins to rise marks the end of the hip motion and the beginning of pelvic and spine motion.

The causes of false positive Thomas test include:

  1. Wrong technique is the most common cause,
  2. Fixed pelvic obliquity in scoliosis and polio,
  3. Exaggerated lordosis in obese individuals,
  4. Malformed pelvis.
Thomas Test positive
Positive Thomas Test – Flexion contracture of the left hip

How reliable is the Thomas test?

Neither the original Thomas Test nor the suggested variations have ever been substantiated for reliability, sensitivity, or specificity1:

Modified Thomas Test

A modified thomas test is commonly used to help eliminate the effect of the lumbar curve.

For the modified Thomas Test , the patient is positioned in sitting at the end of the bed. From this position, the patient is asked to lie back, while bringing both knees against the chest. Once in this position, the patient is asked to perform a posterior pelvic tilt. While the contralateral hip is held in maximum hip flexion by the patient’s hands, the tested limb is lowered over the end of the bed toward the floor.

What does a positive modified Thomas Test indicate?

If normal, the thigh should be parallel with the bed, in neutral rotation, and neither abducted nor adducted, with the lower leg being perpendicular to the thigh and in neutral rotation. There should be 100–110 degrees of knee flexion present with the thigh in line with the table.

If the thigh is raised compared to the table, a decrease in the flexibility of the iliopsoas muscle complex should be suspected.

If the rectus femoris is adaptively shortened, the amount of knee extension should increase with the application of overpressure into hip extension.

If the decrease in flexibility lies with the iliopsoas, attempts to correct the hip position should result in an increase in the external rotation of the thigh.

The application of overpressure into knee flexion can also be used. If the increase in knee flexion produces an increase in hip flexion (the thigh rises higher off the bed), the rectus femoris is implicated, whereas if the overpressure produces no change in the degree of hip flexion, the iliopsoas is implicated.

The data illustrated that reliable assessment using the modified Thomas test may be influenced by:

  1. variations in the application of assessment criteria among examiners,
  2. the scoring method used,
  3. the consistency and accuracy of establishing surface landmarks,
  4. and the population from which the sample was selected.
Modified Thomas Test
Modified Thomas Test

Notes

Two things must be remembered when interpreting the results of Thomas Test:

  1. The criteria are arbitrary and have been shown to vary between genders and limb dominance and to depend on the types and the levels of activity undertaken by the individual.
  2. The apparent tightness might simply be normal tissue tension, producing a deviation of the leg because of an increased flexibility of the antagonists.

The principle behind Thomas test:

Fixed saggital plane deformity is compensated by pelvic extension (flexion deformity) and vice versa. This is produced by lumbar lordosis which shares the compensation. The combined effect allows a patient to walk with feet touching the ground in conjunction with knee flexion. While we flex the normal hip the deformity in pelvis first gets corrected (something akin to squaring pelvis) then lumbar lordosis is corrected simultaneously revealing the deformity.

Related Anatomy

Iliopsoas Muscle:

Rectus Femoris Muscle:

Pectineus Muscle:

Gracilis Muscle:

Tensor Fascia Latae Muscle:

Sartorius Muscle:

See Also: Pelvic Anatomy
MuscleOriginInsertionNerve
Iliopsoas muscleTransverse processes of L1-L5 vertebraLesser trochanterFemoral Nerve
The rectus femoris muscleAnterior inferior iliac spine AIIS, acetabular rimPatella and tibial tubercleFemoral Nerve
Pectineus musclePectineal line of pubisPectineal line of femur Femoral and obturator Nerve
Gracillis muscleInferior symphysis/ pubic arch Proximal medial tibiaObturator Anterior Nerve
Tensor fasciae latae muscle (tensor fasciae femoris)Anterior iliac crestIliotibial bandSuperior gluteal Nerve
Sartorius muscleAnterior superior iliac spine ASISProximal medial tibiaFemoral Nerve
The hip flexor muscles

Progressive Fibrosis of the Quadriceps:

Progressive fibrosis of the quadriceps muscle is a condition in which extension contracture of the knee develops in early childhood as a result of fibrosis of one or more components of the quadriceps muscle. The condition is more common in girls than in boys.

The exact cause of progressive fibrosis of the quadriceps is not known. Gunn2 first proposed that it was a sequela of multiple injections of antibiotics into the thigh muscles during early infancy.

The pathophysiology of progressive fibrosis is speculative. It has been proposed that the volume of drug injected in
very young infants compresses the capillaries and muscle fibers and causes muscle ischemia, which leads to fibrotic changes. Local necrosis may occur as a result of focal disruption of fibers at the site of injection. The irritative nature of the injected drug may also play a role in producing fibrosis.

Clinical Symptoms:

  1. The clinical hallmark of progressive fibrosis of the quadriceps is painless, progressive limitation of both active and passive knee flexion with an extension contracture. The vastus intermedius is most commonly involved. Fibrosis
    occurs more distally than proximally, within and between the muscle fibers.
  2. A dimple in the skin may be present because of the rigid, fibrous septa that extend between the skin and the deep fascia; the dimple deepens with forced flexion of the knee.
  3. Range of motion is painless within the available arc.
  4. The involved muscle is atrophic, with subcutaneous hardness and limitation of motion.
  5. Genu recurvatum may develop in severe cases.
  6. The patella is high riding. Habitual dislocation of the patella may occur in chronic cases.
  7. Knee flexion in these patients is accomplished through lateral dislocation of the patella. With the patella held within the groove of the femur, the knee cannot be flexed. In these patients the vastus lateralis is usually involved. This condition differs from congenital lateral dislocation of the patella in that it is an acquired contracture resulting from progressive fibrosis.

Treatment

Two different surgical releases have been advocated for the treatment of quadriceps fibrosis:

  1. The first is surgical release of the extension contracture by proximal division of the fibrotic muscular bands, which is often combined with transverse division of the iliotibial tract. This approach is preferred in patients younger than 10 years in whom no radiographic changes are present in the distal end of the femur.
  2. The other surgical approach is V-Y quadricepsplasty to lengthen the extensor mechanism as a whole when the fibrosis is extensive. Postoperative extensor lag may be present but resolves with time in most cases. The extensor lag is more prevalent following V-Y plasty than after proximal release of the fibrotic bands.

When the fibrosis is chronic and genu recurvatum is present, skeletal changes may develop in the distal end of the femur where the articular surface points anteriorly. In such cases it may be necessary to perform distal femoral flexion osteotomy to gain knee flexion and maintain joint congruity.

Hip Flexion contracture

A flexion contracture at the hip is a common occurrence. Hip flexion contractures can result from:

  1. adaptive shortening of the iliopsoas muscle or rectus femoris muscles;
  2. contracture of the anterior hip capsuloligamentous complex.

These changes to the soft tissue and connective tissues around the hip can result from OA, injury, or sustained postures involving hip flexion. The resulting anterior rotation of the pelvis shifts the weight-bearing of the hip to a thinner region of hyaline cartilage, in both the femur and the acetabulum, and places the hip extensors in a state of low-level tension.

Flexion contractures can be diagnosed using the Thomas test.

The intervention for the contracture is based on the cause. Adaptive shortening of the contractile tissues may be addressed using muscle energy, passive stretching, and myofascial techniques. Stretching of the capsuloligamentous complex is accomplished by grade III distraction mobilizations and by prolonged stretching.

Reference

  1. The modified Thomas test is not a valid measure of hip extension unless pelvic tilt is controlled | Andrew D. Vigotsky, Gregory J. Lehman, Chris Beardsley, Bret Contreras, Bryan Chung, Erin H. Feser PeerJ. 2016; 4: e2325. Published online 2016 Aug 11. doi: 10.7717/peerj.2325 PMCID: PMC4991856.
  2. Gunn DR: Contracture of the quadriceps muscle. A discussion on the etiology and relationship to recurrent dislocation of the patella, J Bone Joint Surg Br 46: 492, 1964.
  3. Clapis, Davis & Davis (2007) Clapis PA, Davis SM, Davis RO. Reliability of inclinometer and goniometric measurements of hip extension flexibility using the modified Thomas test. Physiotherapy Theory and Practice. 2007;24:135–141. doi: 10.1080/09593980701378256.
  4. Harvey D: Assessment of the flexibility of elite athletes using the modified Thomas test. Br J Sports Med 32:68–70, 1998.
  5. Peeler JD, Anderson JE. Reliability limits of the modified Thomas test for assessing rectus femoris muscle flexibility about the knee joint. J Athl Train. 2008 Sep-Oct;43(5):470-6. doi: 10.4085/1062-6050-43.5.470. PMID: 18833309; PMCID: PMC2547866.
  6. Lee LW, Kerrigan DC, Della Croce U. Dynamic implications of hip flexion contractures. Am J Phys Med Rehabil. 1997 Nov-Dec;76(6):502-8. doi: 10.1097/00002060-199711000-00013. PMID: 9431270.
  7. Magee D.J. Orthopedic Physical Assessment. 4th ed. Vol. 2002. Philadelphia, PA: WB Saunders; Hip; pp. 607–660.
  8.  Kendall F.P, McCreary E.K, Provance P.G, Rodgers M.M, Romani W.A. Muscles: Testing and Function, With Posture and Pain. 5th ed. Vol. 2005. Baltimore, MD: Lippincott Williams & Wilkins; Lower extremity; pp. 359–464.
  9. Peeler J, Anderson J.E. Reliability of the Thomas test for assessing range of motion about the hip. Phys Ther Sport. 2007;8(1):14–21.
  10. Cibere J, Thorne A, Bellamy N, Greidanus N, Chalmers A, Mahomed N, Shojania K, Kopec J, Esdaile JM. Reliability of the hip examination in osteoarthritis: effect of standardization. Arthritis Rheum. 2008 Mar 15;59(3):373-81. doi: 10.1002/art.23310. PMID: 18311750.
  11. Clinical Tests for the Musculoskeletal System 3rd Ed. Book.
  12. Mark Dutton, Pt . Dutton’s Orthopaedic Examination, Evaluation, And Intervention, 3rd Edition Book.
  13. Millers Review of Orthopaedics, 7th Edition Book.


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